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Nuclear NFATc1 Causes Development of Fms-like Receptor Tyrosine Kinase 3 Receptor (FLT3) Internal Tandem Duplication Mutation-Positive (FLT3-ITD) Acute Myeloid Leukemia (AML) and Mediates Drug Resistance

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dc.bibliographiccitation.firstpage1722
dc.bibliographiccitation.issue22
dc.bibliographiccitation.journalBlood
dc.bibliographiccitation.lastpage1722
dc.bibliographiccitation.volume128
dc.contributor.authorSolovey, Maria A.
dc.contributor.authorWang, Ying
dc.contributor.authorGattenloehner, Stefan
dc.contributor.authorHessmann, Elisabeth
dc.contributor.authorMetzelder, Stephan
dc.contributor.authorMichel, Christian
dc.contributor.authorEllenrieder, Volker
dc.contributor.authorGoethert, Joachim R.
dc.contributor.authorNeubauer, Andreas
dc.contributor.authorBurchert, Andreas
dc.date.accessioned2020-12-10T18:38:47Z
dc.date.available2020-12-10T18:38:47Z
dc.date.issued2016
dc.identifier.doi10.1182/blood.V128.22.1722.1722
dc.identifier.eissn1528-0020
dc.identifier.issn0006-4971
dc.identifier.urihttps://resolver.sub.uni-goettingen.de/purl?gro-2/77436
dc.language.isoen
dc.notes.internDOI Import GROB-354
dc.titleNuclear NFATc1 Causes Development of Fms-like Receptor Tyrosine Kinase 3 Receptor (FLT3) Internal Tandem Duplication Mutation-Positive (FLT3-ITD) Acute Myeloid Leukemia (AML) and Mediates Drug Resistance
dc.typejournal_article
dc.type.internalPublicationyes
dspace.entity.typePublication

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